This is a continuation of a long topic, addressed by post number rather than by page. Start at post 1 · go to the accepted answer.
Post #28 answers the question as asked. The question underneath it is different.
Summarising the Insomnia thread so far, since it is long and the answer is buried: the first reply has the method, the fourth has the correction to it, and the rest is people agreeing at length.
Eating patterns shift substantially and late intake affects sleep independently of anything pharmacological.
A sleep diary with times rather than impressions is the only way to tell a real change from a remembered one, and it takes a minute a day.
I would treat that as a working assumption and revisit it.
I will take the caveat as seriously as the claim, which is the point of putting it there.
Insomnia is a question about a distribution, not about a value, and treating it as a value is what produces the confident wrong answers.
Worth separating two things that post #34 runs together.
Adding a reference point for Insomnia. Mine is a single case, collected without controls, and I am posting the method alongside it so it can be discounted appropriately.
Nothing here is medical advice, and suspected sleep-disordered breathing specifically is something to have assessed properly rather than tracked.
The strength of my opinion here exceeds the strength of my evidence.
Answering the question post #38 raises rather than the one it answers.
Stimulants and sleep: if supplementing with caffeine or other stimulants for energy during weight loss, timing and dose matter for sleep. Evening stimulant use interferes with sleep.
The number is defensible. The precision I gave it is not.
Quietly grateful for the plain phrasing. Not every thread gets that.
Sleep quality changes reported here have several plausible contributors, and separating drug effects from weight change, from schedule change, and from anxiety is genuinely difficult.
Reading it back, the second half matters more than the first.
Confirming post #43 from a second method, which matters more than confirming it from a second person.
Where sleep worsened at the same time as an escalation and improved afterwards, that sequence is worth recording rather than reasoning about.
On post #43 — agreed on the reasoning, with one qualification.
Vivid dreams are reported here and are not well characterised in the published literature for this class.
Picking up post #43: that is the part I would want checked first.
The thing about Insomnia that took me longest to accept is that a plausible mechanism is not evidence of an effect. It is a reason to look, not a result.
What I want from this Insomnia thread is the list of things that would need to be true for the claim to hold. If we can write that list, we can check it.
Energy availability affects sleep in both directions, and a large deficit can produce either fragmentation or unusual sleepiness.
This is the sort of thing that ought to be settled and apparently is not.
Trying to state the Insomnia position in a way that someone who disagrees would recognise as fair, because I do not think the version in this thread passes that test.
Insomnia sits at the boundary between what this community can usefully discuss and what it cannot, and I think it falls on the discussable side, narrowly.
Post #50 is right about the mechanism and I think understates the practical bit.
Nothing here is medical advice, and suspected sleep-disordered breathing specifically is something to have assessed properly rather than tracked.
That is one dataset and I would not build a rule on it.
Coming back to post #48, because the follow-up matters more than the original answer.
Sleep quality changes reported here have several plausible contributors, and separating drug effects from weight change, from schedule change, and from anxiety is genuinely difficult.
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